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A direct correlation was found with HDL and inverse ones were found with triglycerides, insulin, glucose, and HOMAIR. Adjustment for age, gender, race, BMI, and smoking status decreased each of the correlations found but they remained significant (Supplementary Table S1 online). No significant associations were seen with smoking status, physical activity, alcohol intake, caffeine, blood pressure, and total or LDL cholesterol in adjusted and unadjusted analyses. Compared to baseline, participants had lower levels of HMW and total adiponectin at the end of each of the three feeding periods (Table 3). Percent Alisertib ic50 HMW decreased ?1% from baseline for all three diets although this was not statistically significant for any of the diets (P > 0.05). Although both total and HMW adiponectin decreased from baseline after each diet, there were significant differences in the level of decrease selleck chemicals between diets. Of the three diets, a diet rich in unsaturated fat was beneficial in retaining high levels of HMW and total adiponectin. In comparison to the protein diet, the unsaturated fat diet achieved greater levels of HMW (+8.4%, P = 0.005) and total adiponectin (+5.6%, P 0.7 for all tests). A similar pattern was evident in analyses stratified by race or gender. Changes in adiponectin Cefaloridine from the three diets were then compared with changes in cardiovascular risk factors. Six different comparisons could be made (i.e., three baseline-to-diet or three diet-to-diet). In all such comparisons, changes in total adiponectin were significantly correlated with changes in HDL (with Spearman correlations of 0.20�C0.37, all P